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Volume 15, Issue 4, pp. 382-394
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History
- Received July 13, 2016
- Revision received November 16, 2016
- Accepted November 29, 2016
- Published first January 20, 2017.
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- Previous version (January 20, 2017 - 12:00).
- Previous version (March 6, 2017 - 06:25).
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©2017 American Association for Cancer Research.
Author Information
- Lina Y. Dimberg1,
- Christina G. Towers1,
- Kian Behbakht2,
- Taylor J. Hotz1,
- Jihye Kim3,
- Susan Fosmire4,
- Christopher C. Porter4,
- Aik-Choon Tan3,
- Andrew Thorburn1, and
- Heide L. Ford1,2,*
- 1Department of Pharmacology, University of Colorado Anschutz Medical Campus, Aurora, Colorado.
- 2Department of Obstetrics and Gynecology, Division of Medical Oncology, University of Colorado Anschutz Medical Campus, Aurora, Colorado.
- 3Department of Medicine, Division of Medical Oncology, University of Colorado Anschutz Medical Campus, Aurora, Colorado.
- 4Department of Pediatrics, Division of Pediatric Hematology and Oncology, University of Colorado Anschutz Medical Campus, Aurora, Colorado.
- ↵*Corresponding Author:
Heide L. Ford, University of Colorado School of Medicine, 12800 E 18th Avenue, P18-6115, Mail Stop 8303, Aurora, CO 80045. Phone: 303-724-3509; Fax: 303-724-3663; E-mail: heide.ford{at}ucdenver.edu
L.Y. Dimberg and C.G. Towers contributed equally to this article.
April 2017
Volume 15, Issue 4
Volume 15, Issue 4
A Genome-Wide Loss-of-Function Screen Identifies SLC26A2 as a Novel Mediator of TRAIL Resistance
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